(R,S)-2-(4-이소프로필-4-메틸-5-옥소-2-이미다졸린-2-일)퀴놀린-3-카르복실릭 산

그림문자(GHS)

• 그림문자(GHS)

  

• 신호 어

  Warning

• 유해·위험 문구

  H312：피부와 접촉하면 유해함

• 예방조치문구

  P280：보호장갑/보호의/보안경/안면보호구를 착용하시오.

(R,S)-2-(4-이소프로필-4-메틸-5-옥소-2-이미다졸린-2-일)퀴놀린-3-카르복실릭 산 화학적 특성, 용도, 생산

• 용도

  Agricultural chemical.

• 위험도

  Moderately toxic by ingestion and skin contact. Low toxicity by inhalation.

• Pharmacology

  Plant Metabolism. The selectivity of imazaquin is due to differential rates and routes of metabolism in tolerant crops versus susceptible weeds (3). The primary metabolite in soybeans is a pyrroloquinoline acetamide (41, Fig. 18), which is immobile in the plant and is not an inhibitor of acetolactate synthase. This metabolite is further broken down to eventually yield a dicarboxylic acid.
  Animal Metabolism. Metabolism studies in the rat showed that they rapidly excreted unchanged imazaquin in the urine (5). There was no accumulation of imazaquin or any of its derivatives in the liver, kidney, muscle, fat, or blood. There was no detectable residue in the liver, kidney, muscle, or fat of lactating goats and laying hens, which were fed imazaquin for 7 consecutive days (5).

• 신진 대사

  Imazaquin kills plants by inhibiting acetolactate synthase (ALS) (I50 = 1 μM), which is the first common enzyme in the biosynthesis of the branched chain amino acids, valine, leucine, and isoleucine. Imazaquin is rapidly absorbed through the roots of plants. Once it enters the plant, imazaquin rapidly translocates to the growing points and growth ceases within 1 day after herbicide application followed by chlorosis and then necrosis of the growing points. Total plant death will occur within 2 to 3 weeks after treatment.

(R,S)-2-(4-이소프로필-4-메틸-5-옥소-2-이미다졸린-2-일)퀴놀린-3-카르복실릭 산 준비 용품 및 원자재

(R,S)-2-(4-이소프로필-4-메틸-5-옥소-2-이미다졸린-2-일)퀴놀린-3-카르복실릭 산 공급 업체

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