Overview Application Administration method Mode of action Toxicity and environmental issue References
ChemicalBook > CAS DataBase List > Propiconazole


Overview Application Administration method Mode of action Toxicity and environmental issue References
Propiconazole Structure
  • CAS No.60207-90-1
  • Chemical Name:Propiconazole
  • CBNumber:CB0403173
  • Molecular Formula:C15H17Cl2N3O2
  • Formula Weight:342.22
  • MOL File:60207-90-1.mol
Propiconazole Property
  • Melting point: :<25 °C
  • Boiling point: :180°C (0.1 torr)
  • Density  :1.2700
  • vapor pressure  :5.6 x l0-5 Pa (25 °C)
  • refractive index  :1.6100 (estimate)
  • Flash point: :11 °C
  • storage temp.  : APPROX 4°C
  • form  :neat
  • Water Solubility  :100 mg l-1(2O °C)
  • pka :2.94±0.12(Predicted)
  • Specific Gravity :1.29 (20℃)
  • Merck  :13,7910
  • BRN  :9349305
  • Stability: :Stable. Incompatible with strong oxidizing agents.
  • CAS DataBase Reference :60207-90-1(CAS DataBase Reference)
  • EWG's Food Scores :2-3
  • NCI Dictionary of Cancer Terms :orbit; trust
  • EPA Substance Registry System :Propiconazole (60207-90-1)
Hazard and Precautionary Statements (GHS)
  • Symbol(GHS)
  • Signal wordDanger
  • Hazard statements H225-H301-H311-H331-H370-H302-H317-H330-H410
  • Precautionary statements P210-P260-P280-P301+P310-P311-P301+P312+P330-P304+P340+P310-P403+P233
Propiconazole Price More Price(3)
  • Brand: Sigma-Aldrich
  • Product number: 45642
  • Product name : Propiconazole
  • Purity: mixture of stereo isomers
  • Packaging: 250mg
  • Price: $68.6
  • Updated: 2021/03/22
  • Buy: Buy
  • Brand: Sigma-Aldrich
  • Product number: 45899
  • Product name : Propiconazole solution
  • Purity:  100?ng/μL in methanol, PESTANAL?, analytical standard
  • Packaging: 10ml
  • Price: $112
  • Updated: 2020/08/18
  • Buy: Buy
  • Brand: Cayman Chemical
  • Product number: 18853
  • Product name : Propiconazole
  • Purity: 
  • Packaging: 100mg
  • Price: $25
  • Updated: 2021/03/22
  • Buy: Buy

Propiconazole Chemical Properties,Usage,Production

  • Overview The triazole compound propiconazole (Pcz), 1-[[2-(2,4- dichlorophenyl)-4-propyl-1, 3-dioxolan-2-yl]methyl]-1,2,4-triazole, is a kind of triazole fungicide (Fig. 1). It is used extensively in a variety of applications. It is used on grasses grown for seed, mushrooms, corn, wild rice, peanuts, almonds, sorghum, oats, pecans, apricots, peaches, nectarines, plums and prunes. On cereals it controls diseases caused by Erysiphe graminis, Leptosphaeria nodorum, Pseudocerosporella herpotrichoides, Puccinia spp., Pyrenophora teres, Rhynchosporium secalis, and Septoria spp.[1, 2, 3] .
    Wheat crops are among the commodities most heavily treated with fungicides in the United States, and propiconazole comprises approximately 90% of this application[4]. Propiconazole is soluble in water at a concentration of 110 mg/L, and concentrations as high as 24 mg/L have been reported in waters receiving runoff from agricultural endeavors using the fungicide[5].
    It can act as a potent inhibitor of BR biosynthesis as has been found that it has inhibitory effect on hypocotyl elongation of cress plants (Lepidium sativum)[6]. This inhibitory effect of Pcz was reversed by co-application with brassinolide. Based on the Pcz structure additional BR inhibitors, such as 2RS, 4RS-1-[2-(4- trifluoromethylphenyl)-4-n-propyl-1, 3-dioxolan-2-ylmethyl]-1H- 1,2,4-triazole, were identified[6]. On the other hand, Pcz has been commercially used as fungistat (BannerMaxx, Syngenta) against a broad range of phytopathogenic fungi. Its fungistatic mode of action is the same as that of Ucz and Pac, blocking of lanosterol 14R-demethylase (CYP51A1)[7, 8]. Pcz has also been studied extensively for its toxicity on plants, animals, humans, and the environment[9, 10]. Here we present a molecular genetic analysis of Pcz’s effects on Arabidopsis and maize seedlings.

    Figure 1 the chemical structure of propiconazole
  • Application Triazole fungicides are used as clinical drugs and agricultural pesticides useful for the treatment and protection of corns, fruits, and other plants[11-13]. It is a systemic fungicide with a broad range of activity and a wide range of agricultural cropping applications. It can be used for controlling the fungi diseases caused by Erysiphe graminis; Leptosphaeria nodorum; Pseudocerosporella herpotrichoides; Puccinia spp.; Pyrenophora teres; Rhynchosporium secalis; Septoria spp. It can be used at various plants such as Mushrooms; Corn; Wild rice; Peanuts; Amonds; Sorghum; Oats; Pecan; Fruit including apricots, plums, prunes, peaches & nectarines[14].
  • Administration method For the treatment of Powdery midew of grape, Anthracnose of grape, Anthrax, White rot, spray 25% EC 4000-6000 times mixed with water; For the treatment of Altermaria leaf spot and Venturia inaequalis of apple and pear, spray 25% EC 5000-6000 times mixed with water. For the treatment of Leaf spot and necrosis of peanuts, spray 25% EC 2500-4000 times mixed with water. For the treatment of leaf spot of bananas, spray 25% EC 1500 times mixed with water. For the treatment of Anthrax of Watermelon, Powdery mildew of water-melon, and Leaf spot of watermelon, spray 25% EC 4000-6000 times mixed with water. For the treatment of leaf spot of corn, spray 25% EC 1500 times mixed with water. For the treatment of powdery mildew of wheat, ornamental rust of wheat, spray 25% EC 4000-6000 times mixed with water. For the treatment of Bakanae disease of rice, dip in the seed mixed with water 1000 times for 2 to 3 days[15].
  • Mode of action Propiconazole's mode of action is demethylation of C-14 during ergosterol biosynthesis (through inhibiting the activity of 14a-demethylase as detailed below), and leading to accumulation of C-14 methyl sterols. The biosynthesis of these ergosterols is critical to the formation of cell walls of fungi. This lack of normal sterol production slows or stops the growth of the fungus, effectively preventing further infection and/or invasion of host tissues. Therefore, propiconazole is considered to be fungistatic or growth inhibiting rather than fungicidal or killing [2].
    Sterol 14a-demethylase is a key enzyme for the fungal ergosterol biosynthesis. Inhibition of Sterol 14a-demethylase causes not only depletion of ergosterol but also accumulation of 14-methylsterols in fungal cells [16]. Since 14-methylsterols are unfavourable sterols for bio-membranes, inhibition of 14a-demethylase) seriously impairs the membrane function by the synergistic effects of ergosterol depletion and 14-methylsterol accumulation. Hence, Sterol 14a-demethylase inhibitors are an important class of antifungal agents, and a number of azole derivatives have been put to practical use as the potent antifungal medicines and agrochemicals of this class.
    Propiconazole is also a potent inhibitor of Brassinosteroids biosynthesis. Brassinosteroids (BRs) are poly-hydroxylated steroidal hormones with profound effects on several physiological plant responses. They are involved in regulating cell elongation and division, vascular differentiation, photomorphogenesis, leaf angle inclination, seed germination, stomata development, as well as suppression of leaf senescence and abscission [17-22]. Studies showed that several steps of BR biosynthesis are mediated by cytochrome P450 monooxygenases (P450s) [23]. Triazole compounds have been shown to inhibit P450s, one of the largest and most ubiquitous groups of plant enzymes that catalyze oxidative processes in life systems [24].
  • Toxicity and environmental issue Propiconazole (PCZ) is among the most heavily used in agriculture [25]. Triazole fungicides have a shorter half-life and lower bioaccumulation than organochlorine pesticides, but detrimental effects on the aquatic ecosystem may arise from spray drift or surface run-off after rainfall [11]. They have been reported to undergo transformation to secondary metabolites in terrestrial mammals [12, 26].
    Acute toxicity
    The acute toxicity to mammals for propiconazole technical are an acute oral LD50 for rats of 1,517 mg/kg and 1,344 mg/kg for rabbits. The acute dermal LD50 for rabbit was reported to be >4,000 mg/kg. Propiconazole was considered a slight irritant in rabbit skin and eye irritation studies. A skin sensitization study in guinea pigs demonstrated no allergic effect (2). The acute toxicity to mammals for the formulated products Orbit 3.6E, Tilt 3.6E and Banner 1.1E was as follows: acute oral LD50 for rats of 1,310 mg/kg. The acute dermal LD50 for rabbit was reported to be >5,010 mg/kg. The formulated products were considered a moderate irritant in rabbit skin and eye irritation studies. A skin sensitization study in guinea pigs resulted in the formulated product being considered a sensitizer [2, 27]. EPA toxicologists have recommended that the developmental No-ObservedEffect-Level (NOEL) of 30 mg/kg/day from the rat developmental toxicity study be used for acute dietary risk calculations. The lowest-effect-level (LEL) of 90 mg/kg/day is based on the increased incidence of unossified sternebrae, rudimentary ribs, and shortened or absent renal papillae.
    Chronic effect
    In two-year feeding studies in mice, the NOEL was established at 100 ppm. Significant increases were noted in the incidence of spontaneous liver tumors (benign) observed in male mice at the highest feeding level only. In two-year rat feeding studies, the no-effect-level was established at 100 ppm. There were no tumors in the rat at any feeding level. In one-year feeding studies in dogs, the NOEL was established at 250 ppm, the highest level tested (2). Based on the available chronic toxicity data, EPA has established the RfD for propiconazole at 0.013 mg/kg/day. This RfD is based on a 1 year dog feeding study with a NOEL of 1.25 mg/kg/day and an uncertainty factor of 100. The uncertainty factor of 100 was applied to account for inter-species extrapolation and intra-species variability. Mild irritation of the gastric mucosa was the effect observed at the LEL of 6.2 mg/kg/day [28]. A 21-day subchronic dermal toxicity test in rabbits found after 3 weeks (15 applications) moderate skin irritation to be the only effect following applications of propiconazole at 1000 mg/kg/day [2].
    To fish
    Fish exposed to fungicides in the environment exhibit a variety of biochemical changes, including those in the antioxidant defense system and other biochemical indices [25]. Long-term exposure to PCZ results in significantly increased ROS in fish brain, indicating severe oxidative stress. PCZ induced ROS formation can oxidize most cellular constituents, such as DNA, proteins, and lipids, causing damage to molecules, resulting in reduced enzymatic activity and affecting cellular integrity. Moreover, PCZ can cause inhibition of Na+–K+ATPase in fish brain after long-term exposure probably disturbed the Na+–K+ pump, resulting in the limitation of Na+–K+-ATPase synthesizing capability.
  • References
    1. Worthing, C. R., ed. 1983. The pesticide manual: A world compendium. Croydon, England: The British Crop Protection Council.
    2. Technical Information Bulletin for Propiconazole Fungicide. Ciba-Geigy. Greensboro, NC. 15 pp.
    3. W. T. Thomson. 1997. Agricultural Chemicals. Book IV: Fungicides. 12th edition. Thomson Publications, Fresno, CA.
    4. Garry VF, Schreinemachers D, Harkins ME, Griffith J. 1996. Pesticide appliers, biocides, and birth defects in rural Minnesota. Environ Health Perspect 104:394–399.
    5. Mortensen SR, Johnson KA, Weisskop CP, Hooper MJ, Lacher TE, Kendall RJ. 1998. Avian exposure to pesticides in Costa Rican Banana plantations. Bull Environ Contam Toxicol 60:562–568.
    6. Sekimata K, Han SY, Yoneyama K, Takeuchi Y, Yoshida S, et al. (2002) A specific and potent inhibitor of brassinosteroid biosynthesis possessing a dioxolane ring. J Agric Food Chem 50: 3486 3490.
    7. Yoshida Y, Aoyama Y (1991) Sterol I4a-demethylase and its inhibition: structural considerations on interaction of azole antifungal agents with lanosterol 14a-demethylase (P-45014DM) of yeast. Biochem Soc Trans 19: 778–782.
    8. Wiggins TE, Baldwin BC (1984) Binding of azole fungicides related to dichlobutrazol to cytochrome P450. Pest Sci 14: 206–209.
    9. Li Z, Zlabek V, Velisek J, Grabic R, Machova J, et al. (2011) Multiple biomarkers responses in juvenile rainbow trout, Oncorhynchus mykiss, after acute exposure to a fungicide propiconazole. Environ Toxicol DOI: 10.1002/ tox.20701.
    10. Thorstenson CW, Lode O (2001) Laboratory degradation studies of bentazone, dichlorprop, MCPA, and propiconazole in Norwegian soils. J Environ Qual 30: 947–953.
    11. Konwick BJ, Garrison AW, Avants JK, Fisk AT. 2006. Bioaccumulation and biotransformation of chiral triazole fungicides in rainbow trout (Oncorhynchus mykiss). Aquatic Toxicol 80:372–381.
    12. Chen PJ, Moore T, Nesnow S. 2008. Cytotoxic effects of propiconazole and its metabolites in mouse and human hepatoma cells and primary mouse hepatocytes. Toxicol Vitro 22:1476–1483.
    13. Li ZH, Randak T. 2009. Residual pharmaceutically active compounds (PhACs) in aquatic environment—Status, toxicity and kinetics: A review. Vet Med 52:295–314.
    14. https://sitem.herts.ac.uk/aeru/ppdb/en/Reports/551.htm
    15. http://www.udpf.com/pz2b7acb4-cz69c79f-propiconazole-25-ec.html
    16. Van den Bossche, H. (1985) in Current Topics in Medical Mycology (McCinnis, M. K. ed.), vol.1, pp. 3 13-345, Springer-Verlag, New York
    17. Azpiroz R, Wu Y, LoCascio JC, Feldmann KA (1998) An Arabidopsis brassinosteriod-dependent mutant is blocked in cell elongation. Plant Cell 10: 219–230.
    18. Yamamoto R, Demura T, Fukuda H (1997) Brassinosteroids induce entry into the final stage of tracheary element differentiation in cultured Zinnia cells. Plant Cell Physiol 38: 980–983.
    19. Neff MM, Nguyen SM, Malancharuvil EJ, Fujioka S, Noguchi T, et al. (1999) BAS1: a gene regulating brassinosteroid levels and light responsiveness in Arabidopsis. Proc Natl Acad Sci U S A 96: 15316–15323.
    20. Wada K, Marumo S, Ikekawa N, Morisaki M, Mori K (1981) Brassinolide and homobrassinolide promotion of lamina inclination of rice seedlings. Plant Cell Physiol 22: 323–325.
    21. Sasse JM, Smith R, Hudson I (1995) Effect of 24-epibrassinolide on germination of seeds of Eucalyptus camaldulensis in saline conditions. Proc Plant Growth Regul Soc Am 22: 136–141.
    22. Kim TW, Michniewicz MM, Bergmann DC, Wang ZY (2012) Brassinosteroid inhibits stomatal development by releasing GSK3-mediated inhibition of a MAP kinase pathway. Nature, Epub 2012/02/ 07.
    23. Fujioka S, Yokota T (2003) Biosynthesis and metabolism of brassinosteroids. Annu Rev Plant Biol 54: 137–164.
    24. Mizutani M, Ohta D (2010) Diversification of P450 genes during land plant evolution. Annu Rev Plant Biol 61: 291–315.
    25. Egaas, E., Sandvik, M., Fjeld, E., Kallqvist, T., Goksoyr, A., Svensen, A., 1999. Some effects of the fungicide propiconazole on cytochrome P450 and glutathione Stransferase in brown trout (Salmo trutta). Comp. Biochem. Physiol. C: Toxicol. Pharmacol. 122, 337–344.
    26. Sun, G.B., Thai, S.F., Tully, D.B., Lambert, G.R., Goetz, A.K., Wolf, D.C., Dix, D.J., Nesnow, S., 2005. Propiconazole-induced cytochrome P450 gene expression and enzymatic activities in rat and mouse liver. Toxicol. Lett. 155, 277–287.
    27. Technical Paper. Banner: A Turf Fungicide. Ciba-Geigy. Greensboro, NC. 21 pp.
    28. U.S. Environmental Protection Agency. Propiconazole; Pesticide Tolerances for Emergency Exemptions. Federal Register Document 96-29020. November 12, 1996.
  • Description Propiconazole is a mixture of four stereoisomers and was first developed in 1979 by Janssen Pharmaceutical of Belgium.
  • Chemical Properties normally provided as a yellow liquid
  • Chemical Properties Colorless solid or a yellow thick liquid. Odorless. Commercial product is available as an emulsifi- able concentrate. Physical and toxicological properties may be affected by carrier solvents in commercial formulations.
  • Uses Labelled Propiconazole (P770100). Systemic foliar fungicide. Agricultural fungicide.
  • Uses Agricultural fungicide.
  • Uses Propiconazole is a triazole-based fungicide that is used to control fungi in agriculture, on turf, and in wood.
  • General Description Yellowish odorless liquid. Non corrosive. Used as a fungicide.
  • Reactivity Profile A triazole derivative.
  • Agricultural Uses Fungicide: Used to control fungi on a broad range of crops and turf. Used on ornamentals, range land and rights-of-way to prevent and control powdery mildew and fungi on hardwoods and conifers.
  • Trade name ALAMO®; BANNER®; BENIT®; BREAK®; BUMPER®; CGA-64250®; CGA-92710 F®; DESMEL®; FIDIS®; JUNO®; MANTI® S; MAXX®; NOVEL®; ORBIT®; PRACTIS®; PROPIMAX®; RADAR®; RESTORE®; SPIRE®; STRATEGO® (trifloxystrobin + propiconazole); TASPA®; TILT®; WOCOSIN®
  • Potential Exposure Propiconazole is a triazole fungicide used to control fungi on a broad range of crops and turf. Used on ornamentals, range land and rights-of-way to pre- vent and control powdery mildew and fungi on hardwoods and conifers.
  • Environmental Fate Routes and Pathways, and Relevant Physicochemical Properties Log Kow=3.72
    Solubilities: 47 g l1 in n-hexane; completely miscible with ethanol, acetone, toluene, and octanol; soluble in most organic solvents; in water = 100 mg l-1 at 25°C
    Henry's law constant= 4.1×10-9 atm-cu mmol-1 at 25°C
    Partition Behavior in Water, Sediment, and Soil Terrestrial Fate
    Propiconazole penetrates the terrestrial environment in its function as a fungicide for a variety of crops. In the terrestrial environment, propiconazole is presented to be slightly persistent to persistent. Biotransformation is an important route of transformation for propiconazole, with major transformation products being 1,2,4-triazole and compounds hydroxylated at the dioxolane moiety. Phototransformation on soil or in air is not important for propiconazole transformation. Propiconazole appears to have medium to low mobility in soil. It has the potential to reach ground water through leaching, especially in soils with low organic matter content. Propiconazole is typically detected in the upper soil layers, but transformation products were detected deeper in the soil profile.
    Environmental Persistency
    Propiconazole is persistent and relatively immobile in most soil and aqueous environments. Propiconazole degradation in the aquatic environment appears to be dependent solely on aqueous photolysis. In the soil, propiconazole dissipation appears to be dependent on binding to soil organic matter content. The average half-life in soils ranges from months to a year.
    Propiconazole is expected to biodegrade in the environment. The estimated half-life of propiconazole in aerobic soils is about 40–70 days, and in aerobic waters is about 25–85 days. Based on monitoring data and field tests, propiconazole has an estimated half-life of about 60–96 days in typical soils. No hydrolysis of propiconazole at environmentally relevant pH has been observed.
    Propiconazole degrades into triazole compounds, which may still be toxic. Decomposition of propiconazole by heating may release toxic gases.
  • Metabolic pathway The numerous metabolites of propiconazole are identified from rat urine and feces. Major sites for enzymatic attack are the propyl side chain and the cleavage of the dioxane ring. The 2,4-dichlorophenyl ring is attacked in various ways including the formation of a cyclohexadiene ring system, hydroxylation, replacement of the chlorine substituent by a hydroxy group, and introduction of a methylthio group. The 1,2,4-triazole ring is oxidatively attacked, leading to hydroxy derivatives. The vast majority of the alcoholic and phenolic metabolites are excreted as sulfuric acid and glucuronic acid conjugates. The major metabolic pathway in mice is via cleavage of the dioxane ring. Photolysis causes cleavage of the C1-triazole bond of propiconazole, liberating 1,2,4-triazole as the major product. Six more degradation products are identified which are not included in the mammalian metabolites.
  • Shipping UN3082 Environmentally hazardous substances, liquid, n.o.s., Hazard class: 9; Labels: 9-Miscellaneous hazardous material, Technical Name Required. UN3077 Environmentally hazardous substances, solid, n.o.s., Hazard class: 9; Labels: 9-Miscellaneous hazardous material, Technical Name Required.
  • Degradation Photolysis of propiconazole (1) in hexane and methanolic solutions irradiated with a high-pressure mercury lamp gave a mixture of products 2-8 (Scheme 1) (Dureja et al., 1987). The major product isolated was 1,2,4- triazole (7). A monodechlorinated product (5) was formed in hexane. The dioxolane ring was cleaved in methanol under these conditions.
    Sunlight irradiation on a sandy loam soil surface for 48 days degraded 80% of the applied material and a DTs0 of about 12 days on soil was derived through further rate studies. 1,2,4-Triazole (7) was the major product identified after exposing a thin layer of propiconazole, coated as a thin film, inside a Pyrex flask to sunlight for one month.
  • Toxicity evaluation Propiconazole mode of action is demethylation of C-14 during ergosterol biosynthesis and leading to accumulation of C-14 methyl sterols. The biosynthesis of these ergosterols is critical to the formation of cell walls of fungi. This lack of normal sterol production slows or stops the growth of the fungus, effectively preventing further infection and/or invasion of host tissues. Therefore, propiconazole is considered to be fungistatic or growth inhibiting rather than fungicidal or killing.
  • Incompatibilities The triazoles are sensitive to heat, friction, and impact. Sensitivity varies with the type substitution to the triazole ring. Metal chelated and halogen substitution of the triazol ring make for a particularly heat sensitive material. Azido and nitro derivatives have been employed as high explosives. No matter the derivative these materials should be treated as explosives
  • Waste Disposal Consult with environmental regulatory agencies for guidance on acceptable disposal practices. Contact a licensed disposal facility about surplus and nonrecyclable solutions. Burn in a chemical incinerator equipped with an afterburner and scrubber. Extra care must be exercised as the material in an organic solvent is highly flammable. In accordance with 40CFR165, follow recom- mendations for the disposal of pesticides and pesticide con- tainers. Containers must be disposed of properly by following package label directions or by contacting your local or federal environmental control agency, or by con- tacting your regional EPA office. Incineration or permanga- nate oxidation.
Propiconazole Preparation Products And Raw materials
Raw materials
Preparation Products
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60207-90-1, PropiconazoleRelated Search:
  • 1h-1,2,4-triazole,1-((2-(2,4-dichlorophenyl)-4-propyl-1,3-dioxolan-2-yl)methyl
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  • propiconazole (+)-1-[2-(2,4-dichlorophenyl)-4-propyl-1,3-dioxolan-2-ylmethyl]-1H-1,2,4-triazole
  • Propiconazole E.C.
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  • Propiconazole@1000 μg/mL in Acetone
  • PropiconazolSolution,1000mg/L,1ml
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  • Propiconazole Standard(mixture of isomers)
  • Propuconazol
  • 60207-90-1
  • 50207-90-1
  • C16H17Cl2N3O3
  • C15H17N3O2Cl2
  • C15H17Cl2N3O2
  • C15H10D7Cl2N3O2
  • 2005/48/EU
  • ConazolesMethod Specific
  • PON - PTMethod Specific
  • Alpha sort