염소화 뇌유

그림문자(GHS)

• 그림문자(GHS)

  

• 신호 어

  Danger

• 유해·위험 문구

  H225：고인화성 액체 및 증기

  H370：장기(또는, 영향을 받은 알려진 모든 장기를 명시)에 손상을 일으킴(노출되어도 특정 표적장기 독성을 일으키지 않는다는 결정적인 노출경로가 있다면 노출경로를 기재)

  H411：장기적 영향에 의해 수생생물에 유독함

• 예방조치문구

  P210：열·스파크·화염·고열로부터 멀리하시오 - 금연 하시오.

  P280：보호장갑/보호의/보안경/안면보호구를 착용하시오.

  P370+P378：화재 시 불을 끄기 위해 (Section 5. 폭발, 화재시 대처방법의 적절한 소화제)을(를) 사용하시오.

  P403+P235：환기가 잘 되는 곳에 보관하고 저온으로 유지하시오.

염소화 뇌유 화학적 특성, 용도, 생산

• 화학적 성질

  yellow waxy solid

• 물리적 성질

  Yellow, waxy, nonflammable solid with a chlorine or terpene-like odor. Odor threshold concentration from water is 140 μg/L (quoted, Keith and Walters, 1992).

• 용도

  Toxaphene is an insecticide that contains over 200–700 chemicals and can exist as an yellow to amber solid or gas. Heavily used in the United States until 1982, its use was completely banned in 1990. Toxaphene was used primarily to control insects on cotton crops in the southern United States; it has also been used to control pests on livestock and to control unwanted fish in aquatic environments. Reports have indicated that fish are low metabolizers of toxaphene, whereas other aquatic life such as snails can be extensive metabolizers, thus reducing the toxicity of toxaphene to aquatic life other than fish (Isensee et al., 1979).

• 일반 설명

  Yellow, waxy solid with a pleasant piney odor. Used as an insecticide, primarily for cotton and early growth stages of vegetables. Also peas, soybeans, peanut, corn, and wheat. Not produced commercially in the U.S. since 1982. Only registered for scabies control on cattle in the U.S.

• 공기와 물의 반응

  Insoluble in water.

• 반응 프로필

  TOXAPHENE is decomposed by sunlight and heat. TOXAPHENE is decomposed in the presence of alkali. TOXAPHENE is corrosive to iron. TOXAPHENE is incompatible with strong oxidizers. TOXAPHENE is non corrosive in the absence of moisture.

• 위험도

  Carcinogen.

• 건강위험

  Highly toxic by ingestion; moderately toxicby skin contact and inhalation; may causeskin irritation and allergic dermatitis; causedadverse reproductive effects in experimentalanimals; ingestion of about 1.5–3 g may befatal to adult human; toxic effects in animals include central nervous system stimulation, tremors, convulsions, and liver injury;oral LD50 value (rats): ～100 mg/kg: sufficient evidence of carcinogenicity in animals,causing liver cancer; RCRA Waste Number P123.
  LD50 oral (rat): 50 mg/kg
  LD50 skin (rat): 600 mg/kg
  Studies on the cancer risk assessment oftoxaphene in rodents have shown that itincreased incidence of neoplasms of endocrine organs, thyroid, pituitary, adrenal andmammary glands and reproductive systems(Buranatrevedth 2004).

• 화재위험

  Container may explode in heat of fire. Toxic vapors are generated when heated. Releases hydrochloric acid in the presence of alkali, on prolonged exposure to sunlight, and at temperatures above 311F. Avoid strong oxidizers, corrosive to iron.

• Safety Profile

  Confirmed carcinogen with experimental carcinogenic and tumorigenic data. Human poison by ingestion and possibly other routes. Experimental poison by ingestion, intraperitoneal, and possibly other routes. Moderately toxic experimentally by inhalation and skin contact. Human systemic effects by ingestion and skin contact: somnolence, convulsions or effect on seizure threshold, coma, and allergic skin dermatitis. A skin irritant; absorbed through the skin. Experimental teratogenic and reproductive effects. Human mutation data reported. Liver injury has been reported. Lethal amounts of toxaphene can enter the body through the mouth, lungs, and skin. Systemic absorption of the insecticide is increased by the presence of lgestible oils, and liquid preparations of the insecticide, which penetrate the skin more readily than do dusts and wettable powders.A toxic mixture of organochlorine pesticides stored to some extent in body fat. It resembles chlordane and, to some extent, camphor in its physiological action. It causes diffuse stimulation of the brain and spinal cord resulting in generahzed convulsions of a tonic or clonic character. Death usually results from respiratory failure. Detoxification appears to occur in the liver. The lethal ingestion dose for humans is estimated to be 2-7 g, a toxicity of about four times that of DDT. At least seven human deaths have been reported due to toxaphene, all in chddren. Two families have been made ill by eating vegetables containing a large residue of toxaphene. When heated to decomposition it emits toxic fumes of Cl-.

• Carcinogenicity

  Toxaphene is reasonably anticipated to be a human carcinogen based on sufficient evidence of carcinogenicity from studies in experimental animals.

• 환경귀착

  Soil. Under reduced soil conditions, about 50% of the C-Cl bonds were cleaved (dechlorinated) by Fe2+ porphyrins forming two major toxicants having molecular formulas C10H10Cl8 (Toxicant A) and C10H11Cl7 (Toxicant B). Toxicant A reacted with reduced hematin yielding two reductive dechlorination products (C10H11Cl7), two dehydrodechlo- rination products (C10H9Cl7) and two other products (C10H10Cl6). Similarly, products formed from the reaction of Toxicant B with reduced hematin included two reductive dechlorination products (C10H12Cl6), one dehydrochlorination product (C10H10Cl6) and two products having the molecular formula C10H11Cl5 (Khalifa et al., 1976). The reported dissipation rate of toxaphene from soil is 0.010/day (Seiber et al., 1979).
  Photolytic. Dehydrochlorination will occur after prolonged exposure to sunlight releas- ing hydrochloric acid (U.S. Department of Health and Human Services, 1989). Two compounds isolated from toxaphene, 2-exo,3-exo,5,5,6-endo,8,9,10,10-nonachlor
  Chemical/Physical. Saleh and Casida (1978) demonstrated that Toxicant B (2,2,5- endo,6-exo,8,9,10-heptachlorobornane), the most active component of toxaphene, under- went reductive dechlorination at the geminal dichloro position yielding 2-endo,
  Toxaphene will slowly undergo hydrolysis resulting in the loss of chlorine atoms and the formation of hydrochloric acid (Kollig, 1993). The hydrolysis rate constant for tox- aphene at pH 7 and 25°C was determined to be 8 × 10–6/hour, resulting in a half-life of 9.9 years (Ellington et al., 1987).
  Emits toxic chloride fumes when heated to decomposition (Lewis, 1990).

염소화 뇌유 준비 용품 및 원자재

염소화 뇌유 관련 검색