m-CPBG hydrochloride (80 and 160 nM) significantly reduces water intake elicited by an acute salt load[1].
m-CPBG hydrochloride (third ventricle injection; 160 nM) significantly inhibits water intake in hypovolemic animals[1].
m-CPBG hydrochloride (third ventricle injection; 320 nM) decreases water intake in water-deprived rats[1].
m-CPBG hydrochloride (central administration） inhibits water intake induced by pharmacological activation of central cholinergic and angiotensinergic pathways[1].
| Animal Model: | Wistar male rats[1] |
| Dosage: | 80, 160 and 320 nM |
| Administration: | Ventricle injection |
| Result: | Decreased water intake induced by water deprivation, acute salt load and hypovolemia. |
